Anne Marte Wetting Johansen

• Kværner, Ane Sørlie; Andersen, Astrid Riseth; Henriksen, Hege Berg; Knudsen, Markus Dines; Johansen, Anne Marte Wetting & Hjartåker, Anette [Vis alle 13 forfattere av denne artikkelen] (2023). Associations of the 2018 World Cancer Research Fund/American Institute of Cancer Research (WCRF/AICR) cancer prevention recommendations with stages of colorectal carcinogenesis. Cancer Medicine. ISSN 2045-7634. 12(13), s. 14806–14819. doi: 10.1002/cam4.6119. Fulltekst i vitenarkiv Vis sammendrag

  Background: While adherence to cancer prevention recommendations is linked to lower risk of colorectal cancer (CRC), few have studied associations across the entire spectrum of colorectal carcinogenesis. Here, we studied the relationship of the standardized 2018 World Cancer Research Fund/American Institute for Cancer Research (WCRF/AICR) Score for cancer prevention recommendations with detection of colorectal lesions in a screening setting. As a secondary objective, we examined to what extent the recommendations were being followed in an external cohort of CRC patients. Methods: Adherence to the seven-point 2018 WCRF/AICR Score was measured in screening participants receiving a positive fecal immunochemical test and in CRC patients participating in an intervention study. Dietary intake, body fatness and physical activity were assessed using self-administered questionnaires. Multinomial logistic regression was used to estimate odds ratios (ORs) and 95% confidence intervals (CIs) for screen-detected lesions. Results: Of 1486 screening participants, 548 were free from adenomas, 524 had non-advanced adenomas, 349 had advanced lesions and 65 had CRC. Adherence to the 2018 WCRF/AICR Score was inversely associated with advanced lesions; OR 0.82 (95% CI 0.71, 0.94) per score point, but not with CRC. Of the seven individual components included in the score, alcohol, and BMI seemed to be the most influential. Of the 430 CRC patients included in the external cohort, the greatest potential for lifestyle improvement was seen for the recommendations concerning alcohol and red and processed meat, where 10% and 2% fully adhered, respectively. Conclusions: Adherence to the 2018 WCRF/AICR Score was associated with lower probability of screen-detected advanced precancerous lesions, but not CRC. Although some components of the score seemed to be more influential than others (i.e., alcohol and BMI), taking a holistic approach to cancer prevention is likely the best way to prevent the occurrence of precancerous colorectal lesions.

• Johansen, Anne Marte Wetting; Myhre, Jannicke Borch; Hjartåker, Anette & Andersen, Lene Frost (2019). Validation of energy intake recorded by a 7-day pre-coded food diary against measured energy expenditure in a group of Norwegian adults. PLOS ONE. ISSN 1932-6203. 14(4), s. 1–12. doi: 10.1371/journal.pone.0215638. Fulltekst i vitenarkiv
• Myhre, Jannicke Borch; Johansen, Anne Marte Wetting; Hjartåker, Anette & Andersen, Lene Frost (2018). Relative validation of a pre-coded food diary in a group of Norwegian adults - Comparison of underreporters and acceptable reporters. PLOS ONE. ISSN 1932-6203. 13(8). doi: 10.1371/journal.pone.0202907. Fulltekst i vitenarkiv Vis sammendrag

  Estimating dietary intake is important for both epidemiological and clinical studies. In large studies, a balance has to be achieved between methods with high accuracy and methods that are easy to use. The aim of the present study was to compare results from a pre-coded scanable food diary (PFD) with results from a weighed record (WR) in a group of Norwegian adults. We also explored differences in day-to-day energy intake and the distribution of energy intake across the day in acceptable reporters (ARs) and underreporters (URs). Participants (n = 114, mean age 35 years, 68% women) recorded dietary intake with the PFD for 7 consecutive days. One week after completing the PFD, participants completed a 7 days WR. No difference in mean energy intake was seen between methods. Few differences were seen for the macronutrients, the most noticeable difference being the percentage of energy (E%) from carbohydrates which was significantly lower with the PFD (47 E%) than with the WR (49 E%). For the micronutrients, intakes of calcium and vitamin A were both significantly higher with the PFD than with the WR. Pearson's correlation coefficient ranged from 0.47 (tocopherol) to 0.76 (E% carbohydrates) for all nutrients. Bread intake was significantly lower with the PFD while the intakes of edible fats, cheese and beverages were higher. Twenty-eight percent of the participants were found to be URs with the PFD. No clear pattern of underreporting at certain recording days or times of the day was seen. In conclusion, the results showed similar energy intakes and few differences in food and nutrient intakes between the PDF and the WR at the group level. Somewhat larger differences between the methods were seen at the individual level. Because of the reduced work load on both participants and researchers, the PFD seems a suitable alternative to the WR.

• Apalset, Ellen M; Gjesdal, Clara Gram; Eide, Geir Egil; Johansen, Anne Marte Wetting; Drevon, Christian A & Tell, Grethe S (2010). Dietary vitamins K1, K2 and bone mineral density; The Hordaland Health Study. Archives of Osteoporosis. ISSN 1862-3522. 5(1-2), s. 73–81.
• Boyles, Abee L.; Wilcox, Allen J.; Taylor, Jack A. ; Shi, Min; Weinberg, Clarice R. & Meye, K [Vis alle 22 forfattere av denne artikkelen] (2009). Oral facial clefts and gene polymorphisms in metabolism of folate/one-carbon and vitamin A: a pathway-wide association study. Genetic Epidemiology. ISSN 0741-0395. 33(3), s. 247–255. doi: 10.1002/gepi.20376. Vis sammendrag

  An increased risk of facial clefts has been observed among mothers with lower intake of folic acid or vitamin A around conception. We hypothesized that the risk of clefts may be further moderated by genes involved in metabolizing folate or vitamin A. We included 425 case-parent triads in which the child had either cleft lip with or without cleft palate (CL/P) or cleft palate only (CPO), and no other major defects. We analyzed 108 SNPs and one insertion in 29 genes involved in folate/ one-carbon metabolism and 68 SNPs from 16 genes involved in vitamin A metabolism. Using the Triad Multi-Marker (TRIMM) approach we performed SNP, gene, chromosomal region, and pathway-wide association tests of child or maternal genetic effects for both CL/P and CPO.We stratified these analyses on maternal intake of folic acid or vitamin A during the periconceptional period. As expected with this high number of statistical tests, there were many associations with P-valueso0.05; although there were fewer than predicted by chance alone. The strongest association in our data (between fetal FOLH1 and CPO, P50.0008) is not in agreement with epidemiologic evidence that folic acid reduces the risk of CL/P in these data, not CPO. Despite strong evidence for genetic causes of oral facial clefts and the protective effects of maternal vitamins, we found no convincing indication that polymorphisms in these vitamin metabolism genes play an etiologic role.

• Johansen, Anne Marte W; Wilcox, Allen J; Lie, Rolv Terje; Andersen, Lene F & Drevon, Christian A (2009). Maternal consumption of coffee and caffeine-containing beverages and oral clefts: A population-based case-control study in Norway. American Journal of Epidemiology. ISSN 0002-9262. 169(10), s. 1216–1222. doi: 10.1093/aje/kwp040.
• Johansen, Anne Marte W.; Lie, Rolv T.; Wilcox, Allen J.; Wilcox, Allen; Andersen, Lene Frost & Drevon, Christian A (2008). Maternal dietary intake of vitamin A and risk of orofacial clefts: A population-based case-control study in Norway. American Journal of Epidemiology. ISSN 0002-9262. 167(10), s. 1164–1170. doi: 10.1093/aje/kwn035.

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• Johansen, Anne Marte Wetting; Andersen, Lene Frost & Drevon, Christian A (2007). High maternal dietary intake of vitamin A and risk of orofacial clefts; a population based case-control study in Norway.
• Johansen, Anne Marte Wetting; Andersen, Lene Frost & Drevon, Christian A (2006). Maternal dietary intake of vitamin A and risk of orofacial cleft; a population based case-control study in Norway.
• Johansen, Anne Marte Wetting (2005). Diet, vitamin A and K, genetics and orofacial clefts (OFC).
• Wetting Johansen, Anne Marte (2005). Kost, gener og risiko for leppe-ganespalte. Norsk tidsskrift for ernæring (NTFE). ISSN 1503-5034.
• Fredriksen, Jannicke & Kristensen, Anne Marte (2004). Deklarering av matvarer - er det samsvar mellom deklarerte og analyserte næringstoffverdier? Norsk tidsskrift for ernæring (NTFE). ISSN 1503-5034. 03.
• Kristensen, Anne Marte; Fredriksen, Jannicke; Løken, Elin Bjørge; Borgejordet, Åse & Nordbotten, Astrid (2004). Does nutrition labeling reflect the analyzed contents of processed food items? Vis sammendrag

  Background: Food manufacturers may label their food items to inform consumers about the nutrient composition of the product. According to Norwegian regulations, declared values may originate from one of three sources: (i) analysis of the specific food item, (ii) calculations based on known or actual average values for the ingredients included in the food item or (iii) calculations based on well established and accepted data. Regulations do not require that the manufacturer states which of these sources was used to obtain the declared nutrient values. Aim: The aim of this study was to compare analyzed and declared nutrient values in recently analyzed Norwegian food items. Methods: A total of 121 products were analyzed during 2000-2002, of which 22 were single food items with declared nutrient contents and could therefore be included in the comparison. The analyzed foods represent market leader products within selected food groups and were sampled in the Bergen area as part of the continuous work with the Norwegian Food Composition Table. Analyses of protein (i.e. nitrogen x 6.25), available carbohydrate (sum of starch and mono+disaccharides), and total fat were performed according to standard procedures at the Institute of Nutrition under the Directorate of Fisheries in Bergen. The energy content was calculated based on the analyzed as well as the declared contents of carbohydrate (17 kJ/g), protein (17 kJ/g), and fat (37 kJ/g) and compared with the declared energy value. Results: The table below (not included here) shows the energy content of food items where the declared value differed more than 10 % from the value based on analysis. For poultry products the differences between declared and analyzed fat content explained most of the differences in energy content. No such relationships were found for the other products. Possible explanations for the discrepancies observed include different methods used for analysis, natural variation in the nutrient contents of the ingredients, calculations based on unspecified sources (cf. (ii), (iii) above), and use of different conversion factors when calculating energy content. Calculated energy content based on declared values indicates that the latter explanation did not play an important role. Conclusions: Our findings show differences between analyzed and declared nutrient contents for some foods. Further studies are needed to clarify the possible discrepancies between analyzed and declared nutrient contents. With respect to future nutrition labeling regulations, it is desirable that manufacturers are required to state the source of the declared nutrient contents.

• Kristensen, Anne Marte & Løken, Elin Bjørge (2003). Kan vi stole på matvaretabellen? Norsk tidsskrift for ernæring (NTFE). ISSN 1503-5034. 1(3), s. 8–8.
• Hansen, Lisa Brooke; Myhre, Jannicke Borch; Johansen, Anne Marte Wetting; Paulsen, Mari Mohn & Andersen, Lene Frost (2015). UNGKOST 3. Landsomfattende kostholdsundersøkelse blant elever i 4. -og 8. klasse i Norge, 2015. Folkehelseinstituttet.
• Totland, Torunn Holm; Melnæs, Benedicte Kjerpeseth; Hallén, Ninna; Kigen, Kaja Marie; Lund, Nicolai Andre & Myhre, Jannicke Borch [Vis alle 9 forfattere av denne artikkelen] (2012). Norkost 3 - En landsomfattende kostholdsundersøkelse blant menn og kvinner i Norge i alderen 18-70 år, 2010-11. Helsedirektoratet.

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