Reidun Torp

• Vera Quesada, Cesar Luis; Shreyas, Rao; Torp, Reidun & Eide, Per Kristian (2023). Widespread distribution of lymphatic vessels in human dura mater remote from sinus veins. Frontiers in Cell and Developmental Biology. ISSN 2296-634X. 11. doi: 10.3389/fcell.2023.1228344. Fulltekst i vitenarkiv
• Vera Quesada, Cesar Luis; Shreyas, Rao; Torp, Reidun & Eide, Per Kristian (2023). Immunohistochemical visualization of lymphatic vessels in human dura mater: methodological perspectives. Fluids and Barriers of the CNS. ISSN 2045-8118. 20(1). doi: 10.1186/s12987-023-00426-3. Fulltekst i vitenarkiv
• Åbjørsbråten, Knut Sindre; Skaaraas, Gry Helen Enger Syverstad; Cunen, Celine Marie Løken; Bjørnstad, Daniel Marelius; Binder, Kristin Maria Gullestad & Bojarskaite, Laura [Vis alle 15 forfattere av denne artikkelen] (2022). Impaired astrocytic Ca2+ signaling in awake-behaving Alzheimer’s disease transgenic mice. eLIFE. ISSN 2050-084X. doi: 10.7554/eLife.75055. Fulltekst i vitenarkiv Vis sammendrag

  Increased astrocytic Ca2+ signaling has been shown in Alzheimer’s disease mouse models, but to date no reports have characterized behaviorally induced astrocytic Ca2+ signaling in such mice. Here, we employ an event-based algorithm to assess astrocytic Ca2+ signals in the neocortex of awake-behaving tg-ArcSwe mice and non-transgenic wildtype littermates while monitoring pupil responses and behavior. We demonstrate an attenuated astrocytic Ca2+ response to locomotion and an uncoupling of pupil responses and astrocytic Ca2+ signaling in 15-month-old plaque-bearing mice. Using the genetically encoded fluorescent norepinephrine sensor GRABNE, we demonstrate a reduced norepinephrine signaling during spontaneous running and startle responses in the transgenic mice, providing a possible mechanistic underpinning of the observed reduced astrocytic Ca2+ responses. Our data points to a dysfunction in the norepinephrine–astrocyte Ca2+ activity axis, which may account for some of the cognitive deficits observed in Alzheimer’s disease.

• Syverstad, Gry-Helen Enger; Melbye, Christoffer; Puchades, Maja ; Leung, Siu Yi Doreen; Jacobsen, Øyvind & Shreyas, Rao [Vis alle 9 forfattere av denne artikkelen] (2021). Cerebral Amyloid Angiopathy in a Mouse Model of Alzheimer’s Disease Associates with Upregulated Angiopoietin and Downregulated Hypoxia-Inducible Factor. Journal of Alzheimer's Disease. ISSN 1387-2877. 83(4), s. 1651–1663. doi: 10.3233/JAD-210571. Fulltekst i vitenarkiv
• Henjum, Kristi; Årskog, Vibeke Helen; Jendresen, Charlotte Bille; Fladby, Tormod; Torp, Reidun & Nilsson, Lars (2020). Analyzing microglial-associated Aβ in Alzheimer’s disease transgenic mice with a novel mid-domain Aβ-antibody. Scientific Reports. ISSN 2045-2322. 10(10590). doi: 10.1038/s41598-020-67419-2. Fulltekst i vitenarkiv
• Stahl, Katja; Rahmani, Soulmaz; Prydz, Agnete; Skauli, Nadia; MacAulay, Nanna & Mylonakou, Maria-Niki [Vis alle 13 forfattere av denne artikkelen] (2018). Targeted deletion of the aquaglyceroporin AQP9 is protective in a mouse model of Parkinson's disease. PLOS ONE. ISSN 1932-6203. 13(3). doi: 10.1371/journal.pone.0194896. Fulltekst i vitenarkiv Vis sammendrag

  More than 90% of the cases of Parkinson’s disease have unknown etiology. Gradual loss of dopaminergic neurons of substantia nigra is the main cause of morbidity in this disease. External factors such as environmental toxins are believed to play a role in the cell loss, although the cause of the selective vulnerability of dopaminergic neurons remains unknown. We have previously shown that aquaglyceroporin AQP9 is expressed in dopaminergic neurons and astrocytes of rodent brain. AQP9 is permeable to a broad spectrum of substrates including purines, pyrimidines, and lactate, in addition to water and glycerol. Here we test our hypothesis that AQP9 serves as an influx route for exogenous toxins and, hence, may contribute to the selective vulnerability of nigral dopaminergic (tyrosine hydroxylase-positive) neurons. Using Xenopus oocytes injected with Aqp9 cRNA, we show that AQP9 is permeable to the parkinsonogenic toxin 1-methyl-4-phenylpyridinium (MPP+). Stable expression of AQP9 in HEK cells increases their vulnerability to MPP+ and to arsenite—another parkinsonogenic toxin. Conversely, targeted deletion of Aqp9 in mice protects nigral dopaminergic neurons against MPP+ toxicity. A protective effect of Aqp9 deletion was demonstrated in organotypic slice cultures of mouse midbrain exposed to MPP+ in vitro and in mice subjected to intrastriatal injections of MPP+ in vivo. Seven days after intrastriatal MPP+ injections, the population of tyrosine hydroxylase-positive cells in substantia nigra is reduced by 48% in Aqp9 knockout mice compared with 67% in WT littermates. Our results show that AQP9 –selectively expressed in catecholaminergic neurons—is permeable to MPP+ and suggest that this aquaglyceroporin contributes to the selective vulnerability of nigral dopaminergic neurons by providing an entry route for parkinsonogenic toxins. To our knowledge this is the first evidence implicating a toxin permeable membrane channel in the pathophysiology of Parkinson’s disease.

• Yang, Jing; Zhang, Rui; Shi, Changhe; Mao, Chengyuan; Yang, Zhihua & Suo, Zhenhe [Vis alle 8 forfattere av denne artikkelen] (2017). AQP4 Association with Amyloid Deposition and Astrocyte Pathology in the Tg-ArcSwe Mouse Model of Alzheimer's Disease. Journal of Alzheimer's Disease. ISSN 1387-2877. 57(1), s. 157–169. doi: 10.3233/JAD-160957.
• Nuruddin, Syed; Syverstad, Gry-Helen Enger; Lillehaug, Sveinung; Leergaard, Trygve Brauns; Nilsson, Lars & Ropstad, Erik [Vis alle 9 forfattere av denne artikkelen] (2014). Elevated mRNA-Levels of Gonadotropin-Releasing Hormone and Its Receptor in Plaque-Bearing Alzheimer's Disease Transgenic Mice. PLOS ONE. ISSN 1932-6203. 9(8). doi: 10.1371/journal.pone.0103607.
• Lillehaug, Sveinung; Syverstad, Gry-Helen Enger; Nilsson, Lars; Bjaalie, Jan G.; Leergaard, Trygve Brauns & Torp, Reidun (2014). Brainwide distribution and variance of amyloid-beta deposits in tg-ArcSwe mice. Neurobiology of Aging. ISSN 0197-4580. 35(3), s. 556–564. doi: 10.1016/j.neurobiolaging.2013.09.013.
• Lillenes, Meryl Sønderby; Støen, Mari; Gómez-Muñoz, Marta; Torp, Reidun; Günther, Clara-Cecilie & Nilsson, Lars [Vis alle 7 forfattere av denne artikkelen] (2013). Transient OGG1, APE1, PARP1 and Polβ expression in an Alzheimer's disease mouse model. Mechanisms of Ageing and Development. ISSN 0047-6374. 134(10), s. 467–477. doi: 10.1016/j.mad.2013.09.002. Vis sammendrag

  Alzheimer's disease (AD) is a disease of major public health significance, whose pathogenesis is strongly linked to the presence of fibrillar aggregates of amyloid-beta (Aβ) in the aging human brain. We exploited the transgenic (Tg)-ArcSwe mouse model for human AD to explore whether oxidative stress and the capacity to repair oxidative DNA damage via base excision repair (BER) are related to Aβ pathology in AD. Tg-ArcSwe mice express variants of Aβ, accumulating senile plaques at 4–6 months of age, and develop AD-like neuropathology as adult animals. The relative mRNA levels of genes encoding BER enzymes, including 8-oxoguanine glycosylase (OGG1), AP endonuclease 1 (APE1), polymerase β (Polβ) and poly(ADP-ribose) polymerase 1 (PARP1), were quantified in various brain regions of 6 weeks, 4 months and 12 months old mice. The results show that OGG1 transcriptional expression was higher, and APE1 expression lower, in 4 months old Tg-ArcSwe than in wildtype (wt) mice. Furthermore, Polβ transcriptional expression was significantly lower in transgenic 12 months old mice than in wt. Transcriptional profiling also showed that BER repair capacity vary during the lifespan in Tg-ArcSwe and wt mice. The BER expression pattern in Tg-ArcSwe mice thus reflects responses to oxidative stress in vulnerable brain structures.

• Nagelhus, Erlend Arnulf; Amiry-Moghaddam, Mahmood ; Bergersen, Linda Hildegard; Bjaalie, Jan G.; Eriksson, Jens & Gundersen, Vidar [Vis alle 12 forfattere av denne artikkelen] (2013). The glia doctrine: Addressing the role of glial cells in healthy brain ageing. Mechanisms of Ageing and Development. ISSN 0047-6374. 134(10), s. 449–459. doi: 10.1016/j.mad.2013.10.001.
• Benfenati, Valentina; Stahl, Katja; Gomis-Perez, Carolina; Toffanin, Stefano; Sagnella, Anna & Torp, Reidun [Vis alle 11 forfattere av denne artikkelen] (2012). Biofunctional Silk/Neuron Interfaces. Advanced Functional Materials. ISSN 1616-301X. 22(9), s. 1871–1884. doi: 10.1002/adfm.201102310.
• Yang, Jing; Lunde, Lisa Kristina; Nuntagij, Paworn; Oguchi, Tomohiro; Camassa, Laura Maria Azzurra & Nilsson, Lars N. G. [Vis alle 11 forfattere av denne artikkelen] (2011). Loss of Astrocyte Polarization in the Tg-ArcSwe Mouse Model of Alzheimer's Disease. Journal of Alzheimer's Disease. ISSN 1387-2877. doi: 10.3233/JAD-2011-110725.
• Stahl, Katja; Mylonakou, Maria-Niki; Skare, Øivind; Amiry-Moghaddam, Mahmood & Torp, Reidun (2011). Cytoprotective effects of growth factors: BDNF more potent than GDNF in an organotypic culture model of Parkinson's disease. Brain Research. ISSN 0006-8993. 1378, s. 105–118. doi: 10.1016/j.brainres.2010.12.090.
• Nuntagij, Paworn; Oddo, Salvatore; LaFerla, Frank M.; Kotchabhakdi, Naiphininch; Ottersen, Ole Petter & Torp, Reidun (2009). Amyloid Deposits Show Complexity and Intimate Spatial Relationship with Dendrosomatic Plasma Membranes: An Electron Microscopic 3D Reconstruction Analysis in 3xTg-AD Mice and Aged Canines. Journal of Alzheimer's Disease. ISSN 1387-2877. 16(2), s. 315–323. doi: 10.3233/JAD-2009-0962.
• Stahl, Katja; Skare, Øivind & Torp, Reidun (2009). Organotypic cultures as a model of Parkinson's Disease. A twist to an old model. Scientific World Journal. ISSN 1537-744X. 9, s. 811–821. doi: 10.1100/tsw.2009.68. Fulltekst i vitenarkiv Vis sammendrag

  Post importert 12/6-2010. Etterrapporteres ikke./tøf

• Nase, Gabriele; Helm, Paul Johannes; Oguchi, Tomohiro; Nilsson, L. N. G.; Lannfelt, L. & Ottersen, Ole Petter [Vis alle 7 forfattere av denne artikkelen] (2008). Local impact of perivascular plaques on cerebral blood flow dynamics in a transgenic mouse model of Alzheimer's disease. Proceedings of SPIE, the International Society for Optical Engineering. ISSN 0277-786X. 6860. doi: 10.1117/12.762739. Fulltekst i vitenarkiv
• Celsi, Fulvio; Svedberg, M; Unger, C; Cotman, CW; Carri, MT & Ottersen, Ole Petter [Vis alle 8 forfattere av denne artikkelen] (2007). Beta-amyloid causes downregulation of calcineurin in neurons through induction of oxidative stress. Neurobiology of Disease. ISSN 0969-9961. 26. doi: 10.1016/j.nbd.2006.12.022.
• Oddo, S; Caccamo, A; Cheng, D; Jouleh, Bahareh; Torp, Reidun & LaFerla, FM (2007). Genetically augmenting tau levels does not modulate the onset or progression of A beta pathology in transgenic mice. Journal of Neurochemistry. ISSN 0022-3042. 102, s. 1053–1063. doi: 10.1111/j.1471-4159.2007.04607.x.
• Mathisen, Thomas Misje; Nagelhus, Erlend Arnulf; Juoleh, Bahareh; Torp, Reidun; Frydenlund, Didrik Søli & Mylonakou, Maria-Niki [Vis alle 18 forfattere av denne artikkelen] (2006). Postembedding Immunogold Cytochemistry of Membrane Molecules and Amino Acid Transmitters in the Central Nervous System. I Záborszky, L.; Wouterlood, F. & Lanciego, J.L. (Red.), Neuroanatomical Tract Tracing: Molecules, Neurons, & Systems. Springer Science+Business Media B.V.. ISSN 0-387-28941-0. s. 72–108.
• Biskup, S; Moore, DJ; Celsi, Fulvio; Higashi, S; West, AB & Andrabi, SA [Vis alle 16 forfattere av denne artikkelen] (2006). Localization of LRRK2 to membranous and vesicular structures in mammalian brain. Annals of Neurology. ISSN 0364-5134. 60(5), s. 557–569.
• Torp, Reidun; Singh, Preet Bano; Sørensen, Dag Reidar; Dietrichs, Espen & Hirschberg, Henry (2006). Vekstfaktorer som nevroprotektiv behandling ved Parkinsons sykdom? Tidsskrift for Den norske legeforening. ISSN 0029-2001. 126(7), s. 899–901.
• Bernard-Trifilo, JA; Kramar, EA; Torp, Reidun; Lin, CY; Pineda, EA & Lynch, G [Vis alle 7 forfattere av denne artikkelen] (2005). Integrin signaling cascades are operational in adult hippocampal synapses and modulate NMDA receptor physiology. Journal of Neurochemistry. ISSN 0022-3042. 93.
• Zhang, L; Shimoji, M; Thomas, B; Moore, DJ; Yu, SW & Marupudi, NI [Vis alle 11 forfattere av denne artikkelen] (2005). Mitochondrial localization of the Parkinson's disease related protein DJ-1: implications for pathogenesis. Human Molecular Genetics. ISSN 0964-6906. 14, s. 2063–2073.
• Rissman, RA; Poon, WW; Blurton-Jones, M; Torp, Reidun; Oddo, S & Vitek, MP [Vis alle 9 forfattere av denne artikkelen] (2004). Caspase-cleavage of tau is an early event in Alzheimer disease tangle pathology. Journal of Clinical Investigation. ISSN 0021-9738. 114(1), s. 121–130.
• Torp, Reidun; Ottersen, Ole Petter; Cotman, C. W. & Head, E. (2003). Identification of neuronal plasma membrane microdomains that colocalize beta-amyloid and presenilin: implications for APP processing. Neuroscience. ISSN 0306-4522. 120(2), s. 291–300.
• Head, E. & Torp, Reidun (2002). Insights into Abeta and presenilin from a canine model of human brain aging. Neurobiology of Disease. ISSN 0969-9961. s. 1–10.
• Torp, Reidun & Ottersen, Ole Petter (2001). Alzheimers sykdom: er forebygging mulig? DIAforskningsnytt (Oslo). ISSN 0808-1212. 5(4), s. 1–2.
• Burton, Brent P.; McCormick, Bruce H.; Torp, Reidun & Fallon, James H. (2001). Three-dimensional reconstruction of neuronal forests. Neurocomputing. ISSN 0925-2312. 38-40, s. 1643–1650.
• Torp, Reidun; Head, E.; Milgram, N. W.; Hahn, F.; Ottersen, Ole Petter & Cotman, C. W. (2000). Ultrastructural evidence of fibrillar beta-amyloid associated with neuronal membranes in behaviorally characterized aged dog brains. Neuroscience. ISSN 0306-4522. 96, s. 495–506.
• Torp, Reidun; Head, E.; Milgram, N. W.; Hahn, F.; Ottersen, Ole Petter & Cotman, C. W. (2000). Ultrastructural evidence of fibrillar beta-amyloid associated with neuronal membranes in behaviorally characterized aged dog brains. Neuroscience. ISSN 0306-4522. 96, s. 495–506.
• Torp, Reidun; Head, E. & Cotman, C. W. (2000). Ultrastructural analyses of b-amyloid in the aged dog brain: Neuronal b-amyloid is localized to the plasma membrane. Progress in Neuro-psychopharmacology and Biological Psychiatry. ISSN 0278-5846. 24, s. 801–810.
• Bassem, Y. A.; Head, E.; Ibrahim, M. A.; Torp, Reidun; Tenner, A. J. & Kim, R. C. [Vis alle 8 forfattere av denne artikkelen] (2000). Molecular dating of senile plaques in the brains of individuals with Down syndrome and in aged dogs. Experimental Neurology. ISSN 0014-4886. 163, s. 111–122.
• Guthman, A.; Fritschy, J.-M.; Ottersen, Ole Petter; Torp, Reidun & Herbert, H. (1998). GABA, GABA transporters, GABA(A) receptor subunits, and GAD mRNAs in the rat parabrachial and Kolliker-Fuse nuclei. Journal of Comparative Neurology. ISSN 0021-9967. 400, s. 229–243.
• Nagelhus, Erlend Arnulf; Veruki, M. L.; Torp, Reidun; Haug, Finn-Mogens Smejda; Laake, Jon Henrik & Nielsen, S.P. [Vis alle 8 forfattere av denne artikkelen] (1998). Aquaporin-4 water channel protein in the rat retina and optic nerve: polarized expression in Mûller cells and fibrous astrocytes. Journal of Neuroscience. ISSN 0270-6474. 18, s. 2506–2519.
• Akbar, M. T.; Torp, Reidun; Danbolt, Niels Christian; Levy, Line Margrete; Meldrum, B. S. & Ottersen, Ole Petter (1997). Expression of glial glutamate transporters GLT-1 and GLAST is unchanged in the hippocampus in fully kindled rats. Neuroscience. ISSN 0306-4522. 78(2), s. 351–359.
• Torp, Reidun; Hoover, Frank; Danbolt, Niels Christian; Storm-Mathisen, Jon & Ottersen, Ole Petter (1997). Differential distribution of the glutamate transporters GLT1 and rEAAC1 in rat cerebral cortex and thalamus: an in situ hybridization analysis. Anatomy and Embryology. ISSN 0340-2061. 195, s. 317–326.
• Ottersen, Ole Petter; Chaudhry, Farrukh Abbas; Danbolt, Niels Christian; Laake, Jon Henrik; Nagelhus, Erlend Arnulf & Storm-Mathisen, Jon [Vis alle 7 forfattere av denne artikkelen] (1997). Molecular organization of cerebellar glutamate synapses. Progress in Brain Research. ISSN 0079-6123. 114, s. 97–107.
• Bjørås, Magnar; Gjesdal, O.; Erickson, J.; Torp, Reidun; Ottersen, Ole Petter & Storm-Mathisen, Jon [Vis alle 8 forfattere av denne artikkelen] (1996). Cloning and expression of a neuronal rat brain glutamate transporter. Brain Research. Molecular Brain Research. ISSN 0169-328X. 36, s. 163–168.
• Ottersen, Ole Petter; Laake, Jon Henrik; Reichelt, W; Haug, Finn-Mogens Smejda & Torp, Reidun (1996). Ischemic disruption of glutamate homeostasis in brain: quantitative immunocytochemical analyses. Journal of Chemical Neuroanatomy. ISSN 0891-0618. 12, s. 1–14.
• Torp, Reidun; Lekieffre, D.; Levy, Line Margrete; Haug, Finn-Mogens Smejda; Danbolt, Niels Christian & Meldrum, B. S. [Vis alle 7 forfattere av denne artikkelen] (1995). Reduced postischemic expression of a glial glutamate transporter, GLT1, in the rat hippocampus. Experimental Brain Research. ISSN 0014-4819. 103(1), s. 51–58.
• Torp, Reidun; Danbolt, Niels Christian; Babaie, Eshrat; Bjoras, M.; Seeberg, Erling Christen & Storm-Mathisen, Jon [Vis alle 7 forfattere av denne artikkelen] (1994). Differential expression of two glial glutamate transporters in the rat brain: an in situ hybridization study. European Journal of Neuroscience. ISSN 0953-816X. 6(6), s. 936–942.
• Storm-Mathisen, Jon; Danbolt, Niels Christian; Rothe, F.; Torp, Reidun; Zhang, N. H. & Aas, J. E. [Vis alle 9 forfattere av denne artikkelen] (1992). Ultrastructural immunocytochemical observations on the localization, metabolism and transport of glutamate in normal and ischemic brain tissue. Progress in Brain Research. ISSN 0079-6123. 94, s. 225–241.
• Ottersen, Ole Petter; Storm-Mathisen, Jon; Bramham, C. R.; Torp, Reidun; Laake, Jon Henrik & Gundersen, Vidar (1990). A quantitative electron microscopic immunocytochemical study of the distribution and synaptic handling of glutamate in rat hippocampus. Progress in Brain Research. ISSN 0079-6123. 83, s. 99–114.
• Bramham, C. R.; Torp, Reidun; Zhang, N. H.; Storm-Mathisen, Jon & Ottersen, Ole Petter (1990). Distribution of glutamate-like immunoreactivity in excitatory hippocampal pathways: a semiquantitative electron microscopic study in rats. Neuroscience. ISSN 0306-4522. 39(2), s. 405–417.
• Ottersen, Ole Petter; Torp, Reidun & Storm-Mathisen, Jon (1989). New aspects of the pathogenesis of ischemic brain damage. Possible involvement of excitatory amino acids]. Tidsskrift for Den norske legeforening. ISSN 0029-2001. 109(26), s. 2674–2677.

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• Nuntagij, Paworn; oddo, salvatore; LaFerla, Frank M; Kotchabhakdi, Nick; Ottersen, Ole Petter & Torp, Reidun (2011). Electron Microscopic 3D Reconstruction Analysis of Amyloid Deposits in 3xTg-AD Mice and Aged Canines. IOS Press. ISBN 978-1-60750-732-1. 1(1). 13 s.

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• Torp, Reidun (2022). Et brytningspunkt i forståelsen av Alzheimers sykdom.
• Syverstad, Gry-Helen Enger; Nuruddin, Syed; Lillehaug, Sveinung; Leergaard, Trygve B.; Haraldsen, Ira Hebold & Torp, Reidun (2013). Targeting gonadotroping-releasing hormone in Alzheimer’s disease.
• Syverstad, Gry-Helen Enger; Lillehaug, Sveinung; Nilsson, Lars N. G.; Bjaalie, Jan G.; Leergaard, Trygve B. & Torp, Reidun (2013). Spatial distribution and variance of amyloid-beta load in middle-aged transgenic Alzheimer’s disease mice with both the Artic and Swedish AβPP mutations.
• Stahl, Katja; WEDEMEYER, MA; FERGUSON, D; NISTOR, GI; HARNESS, J & OKANO, H [Vis alle 9 forfattere av denne artikkelen] (2011). Human induced pluripotent stem cell lines are capable of producing differentiated products of high purity.
• Torp, Reidun (2011). Alzheimer´s sykdom; hva gjør forskerene?
• Torp, Reidun (2011). Understanding plaque formation in models ofAlzheimer disease by use of microscopic techniques.
• Torp, Reidun (2011). How to understand Alzheimer disease through microscopes.
• Torp, Reidun (2011). Alzheimer på cellenivå.
• Jelsness-Jørgensen, Lars-Petter; Bernklev, Tomm; Henriksen, Magne; Torp, Reidun & Moum, Bjørn (2010). Chronic fatigue (CF) is 2-3 times more common in Norwegian IBD patients than the normal population (NP) and it significantly decreases the patients health-related quality of life (HRQOL). Scandinavian Journal of Gastroenterology. ISSN 0036-5521. 45, s. 50–51.
• Torp, Reidun; Jouleh, B; Oddo, S & LaFerla, FM (2004). Ultrastructural analysis of triple-transgenic model of Alzheimer's Disease.
• Torp, Reidun; Jouleh, B; Margol, L; Keyed, R; Head, E & Glabe, C (2004). Soluble Oligomers of Beta-amyloid within human brain.
• Preet, Bano; Hirschberg, Henry; Sørensen, Dag R.; Singh, Narpinder; Dietrichs, Espen & Torp, Reidun (2003). Starting an experimental Parkinsonism project: evalution of 6-OHDA lesions of the nigrostriatal pathway monitored by immunohistochemistry. Nevrodagene 2003..
• Torp, Reidun; Head, E.; Cotman, C. W. & Ottersen, Ole Petter (1999). A-b42 is associated with the plasma membrane of cortical neurons in aged canines.
• Guthman, A.; Fritschy, J.-M.; Ottersen, Ole Petter; Torp, Reidun; Feil, R. & Herbert, H. (1996). GABA, GAD AND GABAA-RECEPTORS IN THE RAT PARABRACHIAL COMPLEX.
• Torp, Reidun; Danbolt, Niels Christian; Storm-Mathisen, Jon & Ottersen, Ole Petter (1996). DIFFERENTIAL CELLULAR AND REGIONAL DISTRIBUTIONS OF THE GLUTAMATE TRANSPORTERS GLT1 AND REAAC1: AN IN SITU HYBRIDIZATION ANALYSIS IN THE RAT BRAIN.
• Eide, Anne Lill & Torp, Reidun (1996). IMMUNOCYTOCHEMICAL CHARACTERIZATION OF HOFMANN NUCLEI NEURONS.

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